Creation-Evolution Headlines
October 2004
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The world view of the scientist, even the most atheistic scientist, is that essentially of monotheism.  It is a belief which is accepted as an article of faith that the universe is ordered in an intelligible way.  Now you couldn’t be a scientist if you didn’t believe these two things... that is a theological position; it’s absolutely clear when you look at history [that it] comes from that theological world view.”
—Paul Davies, theoretical physicist, in the Q&A portion of the new film The Privileged Planet.
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“Evolution Stories Are Subtle and Complex” – Truth or Euphemism?    10/29/2004
A worm brain has photoreceptors similar to those in humans.  What does it mean?  Elizabeth Pennisi in Science1 sets the stage, commenting on work by Arendt et al. in the same issue,2 “Ciliary Photoreceptors with a Vertebrate-Type Opsin in an Invertebrate Brain.”  One might think this demonstrates common ancestry, but Pennisi explains that it’s not a simple evolutionary story:
Despite incredible variation in size and shape, eyes come in just two basic models.  The vertebrates’ photoreceptor cells, typified by rods and cones, are quite distinctive from the invertebrates’.  And although both use light-sensing pigments called opsins, the opsins are quite different in their amino acid makeup.
    For years biologists have argued about how these varied components came to be.  Some insist that eyes evolved only once, despite this modern difference [sic].  Others have argued that optical structures evolved at least once in invertebrates and again in vertebrates.
    New data showing unexpected similarities between photoreceptors of a marine worm and humans add a new twist to this debate.  Detlev Arendt and Joachim Wittbrodt, developmental biologists at the European Molecular Biology Laboratory (EMBL) in Heidelberg, Germany, and their colleagues have found that in addition to its regular opsin pigment, the worm contains another one almost identical to the human’s.  Their finding suggests that even the earliest animals had the makings of both vertebrate and invertebrate visual systems, and that some of the photoreceptor cells in the invertebrate brain were transformed over a series of steps into vertebrate eyes [sic].  Although some researchers are skeptical, others think the data are sound....
  (Emphasis added in all quotes.)
The research team thinks this “sheds new light on vertebrate eye evolution,” but the problem is that it pushes the origin of sight, a complex interaction of multiple functional parts, even farther back in time.  Another problem is that the human-like opsin in the worm has been conserved (unevolved) for 500 million years, according to the standard evolutionary time scale:
Arendt and Wittbrodt jumped into the fray over eye evolution after Arendt noticed some odd cells in the brains of ragworms, a relic marine annelid species that’s been relatively unchanged for the past 500 million years.  “We were surprised,” Arendt recalls, as these cells looked very much like rods and cones.
Further molecular and genetic studies showed that “Not only the morphology [outward appearance] but also the molecular biology of the two types of receptors was already set in our common ancestor” [sic], according to a French biologist.  To put this new discovery into an evolutionary context, Arendt et al. had to invent a hypothetical ancestor even further back in time from the hypothetical ancestor of vertebrates and invertebrates, dubbed Urbilateria: 
They go further to suggest that the two types likely [sic] arose [sic] in a predecessor of Urbilateria.  In that organism, they speculate, the gene for one opsin and the genes to build the one type of photoreceptor cell were duplicated.  The extra set of genes might have evolved [sic] into a different visual system: “We think both photoreceptor cells track back to one cell type,” [Joachim] Wittbrodt [one of the authors of the paper] says.
As the authors put it in conclusion, “The vertebrate eye thus represents a composite structure, combining distinct types of light-sensitive cells with independent evolutionary histories” [sic].  So although this proposal seems to favor those who argue for the single origin of eyes, it illustrates that “evolution stories are subtle and complex.
1Elizabeth Pennisi, “Worm’s Light-Sensing Proteins Suggest Eye’s Single Origin,”
Science, Vol 306, Issue 5697, 796-797, 29 October 2004, [DOI: 10.1126/science.306.5697.796a].
2Arendt et al., “Ciliary Photoreceptors with a Vertebrate-Type Opsin in an Invertebrate Brain,” Science, Vol 306, Issue 5697, 869-871, 29 October 2004, [DOI: 10.1126/science.1099955].
“A likely story” can have opposite meanings depending on the tone of voice.  Is the phrase, “subtle and complex” descriptive of a truth, or a euphemism for a dodge?  Suppose you were a teacher, and your student’s story about the origin of his term paper, which was clearly a hodgepodge of plagiarisms from several internet sources, he described as “subtle and complex.”  Suppose a politician described his flipflops over the years as being subtle and complex.  Suppose your husband’s disastrous room addition project was defended with a story he said was subtle and complex.  One thing is clear about this evolutionary story, as admitted by Pennisi: it is not simple and straightforward.
  The PBS Evolution series tried to claim in 2001 that the eye followed a simple and straightforward progression from simple to complex, using the visual power of suggestion that a series of pictures of animal eyes in a progression from apparently simple to complex suggested an ancestral relationship.  Evolutionists love the word “suggest”.  Scattered similarities between distant organisms, all thriving in their own environments, all using highly-complex functional systems only “suggest” an evolutionary story when you have put yourself under Charlie’s spell and have opened yourself up to the power of suggestion.  Snap out of it.
    Read this evolutionary story with the wide-awake understanding that opsins are very complex proteins (see 10/01/2004 headline and Michael Behe’s Darwin’s Black Box, pp. 15-25).  But their complexity alone is useless without even more complex organs and neurons that can interpret their responses.  Evolutionists want to hypnotize us into the suggestion that stories relating worms to humans by common ancestry are scientific.  Be a clear-headed judge of the evidence.  When missing links have to be invented out of thin air, and when complex functions have to be presumed to have “arisen” [a miracle word] earlier than previously believed, the burden of proof is on the storyteller that the statement, “evolution stories are subtle and complex,” is not just pulling wool over the eyes. 
Next headline on: Darwinism and Evolutionary Theory. • Next headline on: Bugs, Crawlers, and Lower Life Forms.
Platypus Has 10 Sex Chromosomes    10/29/2004
The duck-billed platypus has thrown another curveball at evolutionary theory.  Long puzzling to phylogenists for its mosaic of features that make it seem part mammal, bird and reptile, it has now revealed a genome with 10 sex chromosomes – 10 X in the female, and 5 X plus 5 Y in the male.  Moreover, the sequence of the X chromosomes differs in length and makeup.  The purpose of this arrangement is unclear, but observations by Australian scientists showed that the chromosomes segregate faithfully during meiosis.  “It’s hard to speculate on how that could have evolved,” said one researcher, according to
Nature Science Update.    Whatever the reason, it works.  Science Now says, “the platypus manages to keep its reproduction from going awry.”
For a creationist view on how this is problematic for evolution, see Brad Harrub’s analysis on Apologetics Press.
Nature Science Update relates the Darwinian tale as, “Monotremes were the first group to branch off after mammals evolved [sic] 210 million years [sic] ago.  Their egg-laying shares a common origin [sic] with birds and reptiles, although the bill is thought to have evolved independently.”  Any proof?  No.  Any transitional forms?  No.
    One researcher said, “Mammals are pretty boring when it comes to sex chromosomes.  The platypus is a huge exception.”  Who said sex was boring?  It keeps the Darwin theorists awake at night, but with more pain than pleasure.
Next headline on: Mammals. • Next headline on: Darwinism and Evolutionary Theory.
Will it Become Unlawful to Think Critically?    10/29/2004
The legality of disclaimers in biology textbooks will be decided by a U.S. District Court in Atlanta, Georgia.  A judge will hear the case Nov. 8 about stickers the Cobb County school board put in the books that say, “This textbook contains material on evolution.  Evolution is a theory, not a fact, regarding the origin of living things.  This material should be approached with an open mind, studied carefully and critically considered.”
    Lawyers who sued the school board over these stickers claim that the statement “restricts the teaching of evolution, promotes and requires [sic] the teaching of creationism and discriminates against particular religions.” (Emphasis added.)
The claim that the sticker restricts the teaching of evolution, requires the teaching of creationism or discriminates against particular religions should be approached with an open mind, studied carefully and critically considered, unless the courts make it against the law to do so, at which time all students must assimilate into the Charlie Borg.
Next headline on: Schools.
Cassini Eyes Titan at Close Range    10/28/2004
The
Cassini spacecraft got its closest ever look at Titan on Oct. 26, and downloaded its cargo of precious bits in spite of the rain in Spain and California.  This was the first of 45 targeted flybys of Titan planned during its four-year tour, and should prove one of the best.  The raw and processed images can be viewed on the Cassini website.
    The instrument-laden spaceship had been relatively quiet since its successful orbit insertion last June 30 (see 07/01/2004 headline) and distant untargeted pass of Titan the next day (see 07/06/2004 headline).  Now, after the first of its long, elliptical orbits, it has returned, and what a comeback: passing less than 750 miles above Titan’s haze-shrouded surface, Cassini took unprecedented measurements of Titan and its atmosphere in a whole spectrum of wavelengths – radio, infrared, visible and ultraviolet.
    A press conference unveiling first processed images was held the morning after the flyby.  At the second press conference on Oct. 28, JPL Director and Radar Scientist Dr. Charles Elachi unveiled the first of many radar tracks to come over the four-year mission.  Over the next four years, Cassini radar should map about 25% of the globe at high resolution.  The first radar track showed a complex terrain with possible evidence of lakes; visible-light photographs, however, have not yet seen the glint of reflected sunlight expected if large bodies of liquid were present.  Other images showed streaks due possibly to winds, and organic patches with different thermal properties than those of ice.  No impact craters have been found yet.  The scientists are wondering if any are buried under organic deposits.  Evidence suggests that acetylene, which would be solid at the surface temperatures, is as abundant as ethane.  Some hydrocarbon surface deposits may be flaky, others sticky.
    The ion and neutral mass spectrometer (INMS) found evidence of complex hydrocarbons high in the atmosphere, including diacetylene and benzene, much higher than expected.  This suggests the atmosphere is well mixed.  The INMS principal investigator estimated based on nitrogen isotope ratios that, despite a denser atmosphere than Earth’s on a body less than half its diameter, Titan has lost 3/4 of its atmosphere over “geologic time.”  Surprisingly, the only methane clouds detected were around the south polar feature, although the report at Space.Com questions this.  It may be too early to tell for sure.
    Interdisciplinary scientist Jonathan Lunine, who has studied Titan for over 20 years, appeared happy to see evidence for a dynamic surface and active atmosphere.  There are very complex geologic processes going on, he said, of enormously diverse types.  All the scientists were baffled by long linear features.  They might be ridges or cracks in a thin crust, like cracks in an eggshell.    Mass/density measurements from orbit show that Titan is about half rock, half ice, with the rock presumably having settled at the core.  This means that Titan’s “bedrock,” if visible, should consist of water ice.  That leaves the possibility that some of the linear and streak features might be cryovolcanic in origin, or consist of ammonia-water flows.  Microwave emissions suggest that some areas may be coated with complex hydrocarbons.  How much is liquid and how much is solid is still unclear.  The rich new data sets elicited new puzzles to investigate during future encounters.
    The Huygens Probe scientists from the European Space Agency were excited to get their first high-resolution glimpses of the landing site: though fuzzy before processing, it looked tantalizing and even had a faint X to mark the spot.  The Huygens team leader judged it an excellent site because of its “diversity” (a politically correct answer, some chuckled).  Cassini had all its instruments aboard the ship turned on, and discoveries about surface features, temperatures, plasma characteristics, magnetic field effects, atmospheric and surface composition, and electrostatic effects should continue to get mined out of the treasure trove of new data.
    Coming up: the Huygens Probe mission begins Christmas eve as the probe separates from the mother ship and begins its solo flight to Titan.  On New Year’s Day 2005, Cassini flies by the enigmatic moon Iapetus at about 40,000 miles.  On January 14, the Huygens Probe parachutes open for the historic two-hour descent and landing on the surface, where the world eagerly awaits the first-ever on-site views of this frigid, alien world soaking in hydrocarbons.  Will it land in an ocean or on a solid surface?  The close flyby today may give the best clues before touchdown in January.
Update 11/09/2004: A press release from JPL shows one of the radar images indicating something may be flowing on the surface.  “Titan’s surface is young,” the report says, a growing opinion from the fresh-looking flows and lack of impact craters.  Scientists are still unsure what to make of the dark and light markings.
As reported before, Titan is the moon to watch (see 10/16/2003 headline).  It has many bizarre features that are sure to fascinate scientists and threaten pet theories.  One of the scientists interviewed, who has been studying Titan for 23 years, said two of the biggest questions are, (1) why does Titan have an atmosphere, and (2) what is the source of its methane?  We know that the methane in Titan’s upper atmosphere breaks down in the presence of sunlight and is lost to space, forming a large hydrogen cloud around the huge moon.  Since it is being depleted, why is there any left, if the moon is over four billion years old?  Is the methane resupplied by volcanic outgassing, are there methane lakes or oceans on the surface that evaporate, or was the current methane supply brought in recently by comets?  Till these historic measurements are analyzed, this is a major puzzle on this alien world.  The methane keeps the nitrogen in the atmosphere in a gaseous state, else the entire atmosphere would collapse to the surface in frozen nitrogen snow.
    Some of the Cassini scientists have said that, when giving public talks about the mission, the question they get asked most often is, “Is there life on Titan?”  Sad.  The question reflects indoctrination of a gullible public in evolutionary assumptions.  Even Neil Cavuto at Fox News fell for it.  What do you expect when all the TV science programs keep using misleading phrases like “the building blocks of life” and “early earth in a deep freeze.”  We expect the Titanians will say, “Nobody alive down here, Mate.”
Next headline on: Solar System.
Movie Sequel   10/27/2004
Exploration Films has done it again: it just released Part III of its popular documentary series on Incredible Creatures That Defy Evolution.  Former evolutionist Jobe Martin and narrator David Hames take viewers on a quest around the world for animals both ordinary and exotic that have design features that challenge evolutionary theory.
These films are a lot of fun to watch.  The producers have hit on a winning formula: amazing facts, a little humor, good visuals, and challenging thoughts.  Jobe Martin bubbles with excitement at each creature: mussels, horses, ostriches and much more.  He not only explains why evolutionary theory cannot account for these animal champs, he shows how their abilities enrich the world and our lives, and has a way to explain complex things in a way even kids can enjoy.  This terrific family film is nearly twice the length of the original.  Get the whole set and turn off the Charlie-worshipping science channels with their endless just-so stories.  You’ll learn facts with these films and have fun watching.
Next headline on: Movies.
Middle Earth in Indonesia?  Fossil “Hobbits” Smash Evolutionary Ring   10/27/2004
Here we go again: another alleged human ancestor fossil that shakes up the evolutionary family tree.  No sooner had
Nature1 announced a little 1-meter tall fossil female “hominin” that the discoverers classified as Homo erectus, that the science news media like MSNBC and the BBC flew into action reporting it as “fossil hobbits”  They seem to have all borrowed National Geographic’s artwork, which appeared so fast they must been tipped off.  The drawing by Peter Schouten shows an upright-walking, naked, dark-skinned male with spear and prey over the shoulder.
    The trouble with the fossil is that it was dated at only 18,000 years.  The bones were soggy and unfossilized in the cave, and they were found on an island far from the Indonesian mainland, with stone tools apparently associated with them.  Having a member of genus Homo so far to the East so late in the timeline is forcing a major revision of the idea that modern humans arrived in Europe much earlier.  “My jaw dropped to my knees” said one researcher upon hearing the date.  Homo erectus were long assumed too primitive to have migrated to an island as distant from the mainland as where the two partial skeletons were found.  Yet surprisingly, local natives have legends about “little people” that lived in the jungle, and the BBC article says Henry Gee (editor of Nature) goes as far as to suggest that descendants of this tribe might be found alive today.
    The small stature of these individuals was a big surprise.  Skull capacity of Homo floresiensis, as it was named, is only 380cc – yet evidence of stone tools, upright posture and other “derived” (i.e., advanced) characteristics seemingly contradicts the suggestion these were primitive.  Maybe it’s not brain volume but complexity that matters; after all, DNA can store 1018> bits of information in one cubic millimeter.  “The whole idea that you need a particular brain size to do anything intelligent is completely blown away by this find,” remarked Henry Gee.  Everyone seems to be agreeing on one thing: this astonishing find is going to rewrite the textbooks on human evolution – again.
    Carl Wieland has written a creationist perspective on this find at Answers in Genesis.
Update 11/12/2004: Was so-called Homo florensiensis a small but modern human with a skull deformity called microcephaly?  Michael Balter reported in Science3 that two prominent paleoanthropologists think so.  Brad Harrub and Bert Thompson published an article on Apologetics Press that quotes one of them, and argues this skeleton cannot be anything but fully human.  Balter, for now, leaves the controversy unresolved.
1Rex Dalton, “Little lady of Flores forces rethinking of human evolution,” Nature 431, 1029 (28 October 2004); doi:10.1038/4311029a.
2Mirazon and Foley, “Paleoanthropology: Human evolution writ small,” Nature 431, 1043 - 1044 (28 October 2004); doi:10.1038/4311043a.
3Michael Balter, “Paleoanthropology: Skeptics Question Whether Flores Hominid Is a New Species,” Science, Vol 306, Issue 5699, 1116, 12 November 2004, [DOI: 10.1126/science.306.5699.1116a].
People don’t have to be 5 to 6 feet tall to be people.  There are little people and big people today, yet they show an uncanny commonality in average intelligence, sociability, language, and understanding that Francis Schaeffer used to call the “mannishness of man” (i.e., the set of universal distinctives that separate people from the animals).  Calling something a “hominid” or “hominin” is just a word game to make scientists appear to know more than gullible reporters.  Nothing primitive or transitional about these creatures was found; they have all the marks of full humanity.
    National Geographic and its allies in the news media should be excoriated for the racist artwork they published.  With only bones to draw from, they made Mr. Floresiensis have a protruded chimp-like jaw, an ape-like squat nose, and black skin.  Everything else about the drawing looks fully human, including the proportion of skull size to body size, musculature, walking posture and hunting skill.  Any black person, any respecter of black persons, should be outraged not only at the racist overtones of the picture, but the evolutionary spin put on the data.
    Evolutionary paleoanthropology was in such a complete upheaval of confusion already, what’s another skeleton going to hurt?  It’s just making the rubble of evolutionary storytelling bounce at this point.  The only progress to be hoped for is that they will start calling themselves creationists (see 09/23/2004 headline).
Next headline on: Early Man. • Next dumb story.
Bacterial Flagellum Reveals New Structural Complexity   10/27/2004
The bacterial flagellum, the unofficial mascot of the Intelligent Design movement, got more praise from the evolutionary journal Nature this week: Samatey et al.1 analyzed the hook region in detail and found that it is composed of 120 copies of a specialized protein that “reveals the intricate molecular interactions and a plausible switching mechanism for the hook to be flexible in bending but rigid against twisting for its universal joint function.
    Christopher Surridge, commenting on this paper in the same issue,2 adds that this joint must be able to bend up to 90 degrees in a millisecond or less while rotating at up to 300 times per second.  He says that the researchers describe “how they determined the atomic structure of this super-flexible universal joint, and thereby how it achieves such a feat of engineering.”  (Emphasis added in all quotes.)
1Samatey et al., “Structure of the bacterial flagellar hook and implication for the molecular universal joint mechanism,”
Nature 431, 1062 - 1068 (28 October 2004); doi:10.1038/nature02997.
2Christopher Surridge, “Molecular motors: Smooth coupling in Salmonella,” Nature 431, 1047 (28 October 2004); doi:10.1038/4311047b.
The hook region surely appeared to be one of the simplest-looking parts of the complex molecular motor.  Now, even that little item, something that just bends at an angle, is shown to be exquisitely designed, with exacting specifications to allow bending without twisting.  If all the amino acids in this one protein element were not in the right places, the protein would not work.  And if all 120 were not joined together, and were not assembled at the right time and in the right place, the flagellum would be useless.  Inside that hook is an entire highway of molecular trucks that build the propeller (see 06/14/2004 headline).  No wonder Jonathan Wells remarked, “What we find is irreducible complexity all the way down.”
Next headline on: The Cell. • Next headline on: Intelligent Design.
Adult Stem Cells Continue to Work Miracle Cures   10/27/2004
Chalk up two more amazing successes for adult stem cells (not derived from human embryos, like the controversial ES stem cells):
1. Blindness:  The
BBC News reported that stem cells from the back of the eyeball might be able to restore sight to those afflicted with macular degeneration, a leading cause of blindness.
2. Parkinson’s DiseaseEurekAlert reports that scientists at Thomas Jefferson University found a way to convert adult stem cells to dopamine-producing neurons.  This could lead to treatments for Parkinson’s disease and other diseases caused by a lack of dopamine.
Vote for what works: vote for adult stem cell research.  Embryonic stem cell research – having no track record, no investors, and huge ethical concerns (see 10/21/2004 headline) – makes this vote a no-brainer.
Next headline on: Politics and Ethics. • Next headline on: Health.
How Plants Wax Their Leaves   10/27/2004
Plants have a waxy coating on their leaves, some more and some less, a fact many gardeners may notice without much thought.  A recent paper by two plant biologists in Science1 reveals that even this seemingly ordinary feature comes about only through a complex process in plant cells.  The waxy coating, called the cuticle, is composed of three distinct layers including water-resistant wax crystals that are synthesized by epidermal cells.  Burkhard Schulz and Wolf B. Frommer, commenting on research on this subject, note that over 100 transporter genes of a class named ABC have been discovered in plants, some of which carefully move the insoluble wax molecules to the surface.  They describe the process as major effort in transporting a multitude of large, complex molecules.  Their diagram shows a multitude of molecular machines that take part in the construction of the “elaborate structure” of the cuticle.  Yet they assume this cuticle, with its varied and essential functions, and all the machinery required to product it, arose through a “sloppy” evolutionary history:
When plants moved from water to land [sic] 450 million years ago [sic], they needed [sic] to develop [sic] a sealed surface to protect themselves against water loss in the “dry” air environment.  To solve this problem [sic], plants invented [sic] an epicuticular wax layer that covers the entire surface of the plant that is exposed to air.  This protective wax cuticle also serves a multitude of other functions.  Its elaborate micro- and nanostructure prevents water and other particles from sticking to the surface of leaves, keeping them clean and so enhancing their ability to trap light for photosynthesis.  Adhering water droplets and other particles are washed away in a self-cleaning process called the lotus effect.  The wax layer also filters out damaging ultraviolet rays, prevents volatile chemicals and pollutants from sticking to leaves and stems, and protects plants against attack by microbes and herbivores   (Emphasis added in all quotes.)
Schulz and Frommer want to know “what were the evolutionary steps that led to this innovation?”  They figure that early plants somehow co-opted existing transporter machinery for this new function, because the plants needed it:
How did land plants invent [sic] wax secretion?  The genomes of living land plants contain more than 100 ABC transporter genes.  Because transporters seem to be sloppy [sic] with respect to their substrate specificity, it is feasible [sic] that when plants crept out of the water, they turned [sic] a member of the ABC transporter family into a lipid exporter by ensuring that it became localized to a different cellular compartment.  Perhaps this is an example of an evolutionary principle in which sloppiness is transformed into flexibility.
It’s only a suggestion, they end; “Obviously, there is more work to be done....”
1Burkhard Schulz and Wolf B. Frommer, “A Plant ABC Transporter Takes the Lotus Seat,”
Science, Science, Vol 306, Issue 5696, 622-625, 22 October 2004, [DOI: 10.1126/science.1105227].
Stupid Evolution Quote of the Week, easily.  At the rate the Darwin Party is turning up the propaganda, we’re going to have to make this a daily award.  So plants invented something because they needed it when they crawled out of the water onto the land, and used existing machinery that just happened to be in their toolbox.  This is going to sound so stupid to everybody some day, just like it already does to anybody that cleans his ears of Charlie Ear Wax.
Next headline on: Plants. • Next headline on: Darwinism. • Next dumb story.
DNA Coding Multiplies in Complexity   10/27/2004
As if the discovery that DNA is a language translation system was not enough to challenge evolutionary theories, it is becoming increasing clear that DNA is a code operated by another code.  Science on Oct. 221 had a feature on gene regulation, which writer Elizabeth Pennisi termed “Genome’s Second Code.”  She began, “The genome has more than one code for specifying life.  The hunt for the various types of noncoding DNA that control gene expression is heating up.”  Her second article in the same issue2 describes the “fast and furious hunt for gene regulators.”
    In a letter to Nature,3 a team found that some non-coding DNA is not essential to viability.  They deleted megabases of genetic elements from the mouse genome and could not find anything wrong with the mice.  “Some of the deleted sequences might encode for functions unidentified in our screen,” they suggested; “nonetheless, these studies further support the existence of potentially ‘disposable DNA’ in the genomes of mammals.”  Yet how such DNA would arise if it is not vital for survival, or why it would persist if not essential, seems to contradict the principles of Darwinian natural selection.
    In yet another paper in Science,4 Kosak and Groudine argue that genes are organized in a way to take advantage of space.  Thus, the very spacing and placement of genes with respect to one another and to regulatory elements provide a function: “the clustering of coregulated, lineage-restricted genes indicates a functional organization of transcriptomes that define a given cell type.”  (Transcriptome refers to the body of DNA, regulators and enzymes that work together to transcribe a gene into a protein.)
    Yet another paper in Current Biology5 has complexified the story of telomeres, those end caps on DNA strands that keeps them from unraveling.  Another protein regulator has been found to be essential, and it “adds even more complexity to telomere protein interactions,” a subject already more complex than initially thought.
    In short, it is no longer possible to predict gene expression by just looking at the DNA.  Much more is going on to control what genes get turned on and off and in what order.  The controls are appearing more and more like a super-code behind the genetic code.  Pennisi quotes one geneticist who sighs, “The complexity of the genome is much higher than we have defined for the past 20 years.  We have to change our way of thinking.”  (Emphasis added.)
1Elizabeth Pennisi, “Searching for the Genome’s Second Code,”
Science, Vol 306, Issue 5696, 632-635, 22 October 2004, [DOI: 10.1126/science.306.5696.632].
2Elizabeth Pennisi, “A Fast and Furious Hunt for Gene Regulators,” Science, Science, Vol 306, Issue 5696, 635 , 22 October 2004, [DOI: 10.1126/science.306.5696.635].
3Nobrega et al., “Megabase deletions of gene deserts result in viable mice,” Nature 431, 988 - 993 (21 October 2004); doi:10.1038/nature03022.
4Kosak and Groudine, “Gene Order and Dynamic Domains,” Science, Vol 306, Issue 5696, 644-647 , 22 October 2004, [DOI: 10.1126/science.1103864].
5Lorel Colgin and Roger Reddel, “Telomere Biology: A New Player in the End Zone,” Current Biology, Vol 14, R901-R902, 26 October 2004.
Yes, Darwinites, change your way of thinking.  Think intelligent design.  Thinking itself is not even possible without it.  As could be expected, this section is a treasure trove of juicy quotes and findings of design that leave the Darwinites squirming in their naturalistic straitjackets.  They only have themselves to blame.  They were the ones who said they had to be worn.
Next headline on: Genes and DNA. • Next headline on: Intelligent Design.
Did Language Evolve by Natural Selection?   10/27/2004
In the Oct 14 issue of Nature,1 Gary Marcus (Dept. of Psychology, New York University) appears conflicted about how human language arose.  He wants to attribute it to a Darwinian process:
If, as François Jacob famously argued, evolution is like a tinkerer who builds something new by using whatever is close at hand, then from what is the human capacity for language made?
    Most accounts of the evolution of language [sic] have focused on characterizing changes that are internal to the language system.  Were the earliest forms of language spoken or (like sign language) gestured?  Did language arise suddenly?  Or did it emerge [sic] gradually, progressing step by step from a simple one-word ’protolanguage’ (limited to brief comments about the ‘here and now’) into a more complex system that combined individual words into structured meaningful sentences encompassing the future, the past and the possible – as well as the concrete present?....
  (Emphasis added in all quotes.)
Yet in the last sentence, after considering several options of how language might have arisen and developed by natural processes, he cannot help but wonder at the result:
To the extent that the neural or genetic substrates of language and cognition overlap, language should be understood not just as an adaptation selected for effective communication, but also as a darwinian descendant with modification from pre-existing cognitive systems.  Studying how linguistic systems may have descended with modification from cognitive precursors could in turn elucidate [sic] the oft-noted (but never satisfactorily explained) co-morbidity between language disorders and other cognitive impairments, in terms of overlap in genetic and neural machinery.  At the same time, by highlighting how new mechanisms can be built on top of old, we may be able to make better sense of the mystery of how, within a relatively short period of time, with just a relatively small amount of genetic change, humans evolved the amazing gift of speech.

1Gary Marcus, “Concepts: Before the Word,”
Nature 431, 745 (14 October 2004); doi:10.1038/431745a.
Can a schizophrenic psychologist understand human language?  He sees the “amazing gift of speech,” with all its mystery and wonder, but wants to attribute it to a goddess tinkering with parts cobbled from whatever is at hand (shall we name the evolutionary goddess Charlotte, the fairy godmother of Charles Darwin)?  He can’t have it both ways.  Either language is an amazing gift, designed by intelligence, or it is a meaningless end product of a mindless, undirected natural process.  How much evidence does Mr. Marcus have for his speculation about language arising by evolution?  (Dead silence.)
Next headline on: Human Body. • Next headline on: Darwinism and Evolutionary Theory.
Dinosaurs Survived Cold Arctic   10/27/2004
Dinosaurs, ferns and trees grew in Canada’s far north provinces, according to
EurekAlert report from McGill University.  “You wouldn’t expect it, yet dinosaurs and a great variety of plants lived in the High Arctic 240 to 65 million years [sic] ago,” said Hans Larsson, leader of research over two years. 
Who wouldn’t expect it?  Evolutionists.
Next headline on: Dinosaurs.
Disembodied Brain Flies Jet Aircraft    10/25/2004
Researchers at
University of Florida claim to have connected rat brains neurons in a dish to electrodes, which learned to run an F22 flight simulator.
We can’t speak to the validity of this claim or its interpretation, but what stands out in the article is the awe over the computational abilities of the human brain:
“We’re interested in studying how brains compute,” said Thomas DeMarse, the UF professor of biomedical engineering who designed the study.  “If you think about your brain, and learning and the memory process, I can ask you questions about when you were 5 years old and you can retrieve information.  That’s a tremendous capacity for memory.  In fact, you perform fairly simple tasks that you would think a computer would easily be able to accomplish, but in fact it can’t.
    While computers are very fast at processing some kinds of information, they can’t approach the flexibility of the human brain, DeMarse said.  In particular, brains can easily make certain kinds of computations – such as recognizing an unfamiliar piece of furniture as a table or a lamp – that are very difficult to program into today’s computers.
(Emphasis added.)
Hmmmmm... think about your brain.  The brain is thinking about me, and I’m thinking about it.  I’ll have to think about that one for awhile.
Next headline on: Human Body. • Next amazing story.
Was Darwin Wrong?   10/24/2004
One would think National Geographic wants to know, judging from the cover of the November 2004 issue: “Was Darwin Wrong?”  A reader might think the magazine editors, in light of the controversy about evolution sweeping the country, thought it would be timely to engage in a scientific debate about Darwin’s 19th-century theory.  The reader might anticipate seeing an article quoting experts from both sides.  Flip ahead to page 3, and a double-page photo of a fancy pigeon again frames the question, “Was Darwin Wrong?”  Now turn the page, and the debate is over.  The answer, in 250-point bold type, screams: NO.  The subtitle, in 72-point bold type, declares, “The evidence for Evolution [capitalized] is overwhelming.
    The remainder is mop-up work: photos of naked mole rats, Galápagos finches, skeletons of giraffes and whales and flightless birds and orang-utans, an orchid and its pollinating moth, a bulldog, salmon fry, a Cambrian fossil, a Venus flytrap, ants in amber, DNA, bacteria, a chest X-ray and 18 pages of text by David Quammen.  After rehashing a bit of Darwin-Wallace history, he highlights evolutionary evidences from biogeography, paleontology, embryology and morphology.  The story in a nutshell: “Evolutionary theory... is... such a dangerously wonderful and far-reaching view of life that some people find it unacceptable, despite the vast body of supporting evidence” (p. 6, emphasis added in all quotes).
    And who would these people be?  Fundamentalist Christians, ultra-orthodox Jews, Islamic creationists, Hare Krishnas and “millions of adult Americans” suffering from “honest confusion and ignorance.”  To this group, 45% of whom think God created mankind sometime within the last 10,000 years, and another 37% who mix God and Darwin, this article appears targeted.  (Only 12% believe “humans evolved from other life-forms without any involvement of a god.”)  To these unenlightened 82%, many who “have never taken a biology course that dealt with evolution nor read a book in which the theory was lucidly explained,” Quammen writes to fill in the gaps in their knowledge and alleviate their fears.  Evolution is not dangerous, he explains.  On the contrary, “Evolution is a both a beautiful concept and an important one, more crucial nowadays to human welfare, to medical science, and to our understanding of the world [sic] than ever before” (p. 8).
  Perhaps this targeted message is best illustrated on the last page by a picture of a Russian ex-convict who “carries two enduring remnants from his prison time: a Crucifixion tattoo and drug-resistant TB.  He hopes God will help him, but evolution-based science [sic] is what guides [sic] the truth [sic] for an earthly cure.”
    The article refers to anti-evolutionists as “Creationist proselytizers and political activists, working hard to interfere with the teaching of evolutionary biology in public schools” (p. 6), for example, an unnamed traveling lecturer “from something called the Origins Research Association,” whose dinosaur-illustrated flyer offered “free pizza following the evening service” at a local Baptist church.  Quammen smirks, “Dinosaurs, biblical truth, and pizza: something for everybody.”  Presumably, evolutionists will take the pizza.
    No mention was made of the intelligent design movement, nor any living scientist with a Ph.D. who might whisper “yes” to “Was Darwin wrong?”
Update  Jonathan Wells published a critique of the issue for the
Discovery Institute, and Terry Mortenson issued another rebuttal on Answers in Genesis.
NG has provided a valuable article.  For historians, it will illustrate the desperation of the Darwin Party right before their buddha collapsed.  For logicians, it will provide a classic case study on how to promote a failed theory with logical fallacies, selective evidence, spin doctoring and propaganda.  Thank you, National Geographic, for providing this documentation for future researchers.
    This article has it all: fear-mongering, glittering generalities, analogy, straw man, bluffing, sidestepping, card stacking, big lie, half truth, non-sequitur, extrapolation, equivocation, visualization, personification, and everything else in the whole baloney arsenal.  It won’t work any more.  A new generation of discerning students and adults has arisen.  They are no longer intimidated by bluffing and evasion from the Darwin Party propagandists.  Many of them are readers of Creation-Evolution Headlines and follow the debate closely.  It doesn’t work any more to bluff about the “mountainous accumulation of peer-reviewed scientific studies,” because they are mountain climbers: they read the journals.  They know that evolution is only assumed when mentioned, and that the evolutionary storytelling is inversely proportional to the detail in the observations.  (As a result, they never get much of a workout from climbing said mountains.)  They know how to separate observation from interpretation.  This kind of quick lie in passing, for instance, won’t fool today’s informed readers:
Can we see evolution in action?  Can it be observed in the wild?  Can it be measured in the laboratory?
    The answer is yes.  Peter and Rosemary Grant, two British-born researchers who have spent decades where Charles Darwin spent weeks, have captured a glimpse of evolution with their long-term studies of beak size among Galápagos finches.
Sorry, NG, we examined the Grants’ original papers (see 04/26/2002 and 09/03/2004 headlines) and they showed no such thing.  The best they could do was to find a minor variation in beak size among interfertile finches that reversed when the weather changed, and showed no long-term trend; in fact, the Grants admitted that 30 years is far too short to demonstrate any evolutionary trend.  And your own article showed that no clear example of speciation has been observed (p. 30), and that 999 out of 1000 frames in the “film of evolution” are missing links (p. 25).  The only way to get the attention of today’s informed anti-evolutionists is to stop the propaganda tricks, which are ineffective because we filter them out on the front end, and talk real, objective evidence, addressing the best arguments on both sides.  Since you never do this, we assume you can’t.
    Every one of the other evidences in the National Geographic piece has been contradicted by other scientists, or is irrelevant because it fails to address the main issue Darwin claimed, that everything is related by common descent.  No example of natural selection creating new functional information by an undirected natural process was presented without merely assuming evolution did it – somehow, left unexplained.  Can you believe that they would still dredge up the old stuff about the horse series, vestigial organs and embryologic recapitulation as evidences for evolution?  Come on, you guys, this is 2004.  At least they didn’t showcase peppered moths and Haeckel’s embryos.
    The use of 250-point bold font to shout “No” to the question of whether Darwin was wrong can be interpreted either as (1) a patronizing disdain for the intelligence of the average reader of the magazine, as if they cannot judge the validity of evidence but have to be told the answer (“Evolution is a fact, do you hear?  It’s a fact; are you listening to me?”), or (2) positive self-talk to stave off depression, like a coach’s pep talk to a discouraged team facing formidable odds.  It exudes a flavor of, “Darwin was right, wasn’t he?  Wasn’t he?  We’re losing 82-12 in the polls, but ol’ Charlie boy was right, don’t you think so, Tom?”  “Sure, boss.  Charlie was right.  Don’t let those anti-evolutionist PhDs with all their evidence make you question your faith.  Just ignore them and stick to the playbook: finch beaks prove humans had bacteria ancestors.  Show a picture and maybe they’ll go away and stop bothering us.”
    The worst violation in this propaganda piece was a sin of omission: none of the best arguments against Darwinism were addressed or even mentioned, and intelligent design theory was completely ignored.  Poor Mr. Quammen; he probably wrote his piece before neo-Darwinism was falsified last week (see 10/19/2004 headline).  Should we pressure him for an Archaeoraptor-style retraction? (see 09/27/2000 headline).  Why not, if “a trait that’s valuable in a scientist” is “a willingness to admit when he’s wrong”? (p. 31).
    Read the critiques of this article by Jonathan Wells and Terry Mortensen.  Knowledgeable and articulate readers may wish to take National Geographic up on its offer: “...join our forum and share your thoughts on “Was Darwin Wrong?” at nationalgeographic.com/magazine/0411.  And never underestimate the power of a succinct, informed, articulate letter to the editor.
Next headline on: Darwinism and Evolutionary Theory. • Next headline on: Fossils.
Note to Pastors:  Quammen ends with an interview with an evolutionist who said, “I grew up in a conservative church in the Midwest and was not taught anything about evolution.  The subject was clearly skirted.”  Now, this evolutionist gets “a spiritual experience” by pulling up fossils to piece into his evolutionary just-so stories.  “The evidence is there,” he claims; “It’s buried in the rocks of ages.”  Just thought you would find this little anecdote inspiring.
Scientific Supporters of ES Stem Cell Research Fear Future Abuses    10/21/2004
“How would you know if a human brain was trapped in a mouse’s body?”  This frightful and intriguing question opened an article in Nature this week.1  More on that in a minute.
    Last week, in the Oct. 14 issue,2 a Nature editorial on California’s Stem Cell Proposition 71 stated that “the proposal is less of an unalloyed blessing than it seems.”  Though most professional scientists are eager for funds to test embryonic stem cells, Nature feared that the proposition goes overboard.  It amends the state constitution, threatens a state economy that is near insolvency, and promises it will pay for itself, “But it is not clear that these analyses hold water.”  Worst of all, it prevents oversight by the state legislature, expecting the researchers to police themselves.  Surprisingly, Nature supports government oversight of scientific funding.  The NIH and NSF at the federal level, which operate under the scrutiny of Congress, perform a healthy role: “At these agencies, scientific merit is judged almost entirely by the community itself, but Congress ultimately ensures that the public good is paramount.”  No such policing comes with Prop. 71, however, and the money trail looks too tempting:
Proposition 71, in contrast, would introduce a new model for the support of scientific research at the state level that would rely on mere transparency as a guarantee against abuse.  Although public meetings are promised, the oversight committee would consist mainly of people with close ties to the universities, institutes and companies that stand to benefit from the money spent.  Most of the rest are representatives of disease groups.  The committee makes the ultimate funding decisions and will be allowed to modify NIH rules of informed consent and human-subject protection as it sees fit.
    The advocacy of such people as the actor Christopher Reeve – whose untimely death this week deprives biomedical research of one of its most forceful and effective lobbyists – has helped to elevate the promise of embryonic-stem-cell research, sometimes to unrealistic levels.  It is up to the people of California whether they want to approve Proposition 71.  But if they do, researchers must strive to ensure that no funds will be abused, and they must give full consideration to a wide array of ethical concerns.  Anything less risks damaging public trust in science.
  (Emphasis added in all quotes.)
Yet how effective can self-policing by researchers be, when the temptations for grant money, prizes and lucrative pharmaceutical contracts threaten to make ethics take a back seat?  This was the subject of the editorials this week in Nature1 and Science3 about feeble first attempts in Washington to decide what is right or wrong.  The lack of clear guidelines on stem cell research occasioned the question about human brain cells in mice: how would anyone know?  If the researcher feels he has to experiment with chimeras (see
BreakPoint commentary) to find a cure, on what basis will the scientific community claim it is unethical, and how could they stop it?
    Erika Check wrote about prominent biologists debating such questions just in the last few days at the US National Academies, now that California’s Prop. 71 is already on the ballot and appears poised for an easy win, especially since the state’s popular governor, Arnold Schwarzenegger, has endorsed it along with Michael J. Fox and other celebrities.  Since no clear guidelines exist, and no federal policies have the force of law, the scientists have a free rein to create their own consensus about what is ethical.  The vacuum has allowed some already to charge ahead into areas that are blurring the line between human and animal:
Researchers at the meeting agreed on a lot: that the use of human embryonic stem cells to produce a baby should be banned, for example, and that stem-cell researchers should adopt guidelines to reassure the public that their work is ethically sound.  But they differed on how to handle chimaeras, which mix cells and DNA from different species....
    Scientists could even construct a mouse whose entire brain was made of human-derived cells....
The article quotes Irving Weissman of Stanford who is already creating human-mouse chimeras with private funds.  Weissman claims the “yuck factor” is no reason to ban such research.  The fact that the government so far has not taken the lead in establishing guidelines puts the burden on the scientists themselves, but is this the fox guarding the henhouse?  “That leaves a hole for scientists, who are not sure what the law permits them to do, and lack guidance on their work’s impact on public opinion.”  How, then, can they “reassure the public that their work is ethically sound?”
    Speaking for Science,3 Constance Holden provided more details on the meeting of scientists last week in Washington, DC.  The scientists seemed to agree on little more than the need for guidelines.  They admitted that there is no clear distinction between “stem cell research” and “cloning” even among biotech investors, though the public is usually reassured that cloning is bad.  And they could not answer such basic questions as, “what does it mean to accord an early embryo ‘respect’?”  It didn’t help to hear a legal expert confide, “much assisted reproduction is human experimentation in the name of treatment.”  The potential for deceiving a gullible public appears more powerful than ethical concerns, especially from the so-called religious right (see 09/27/2004 headline). 
EurekAlert reported that the UN is also considering talks about the ethics of therapeutic cloning, as ES stem cell research is called.  Dr. Gerald Schatten (U. of Pittsburgh) argues research first, ethics later as he admits that ES stem cells have no track record: “Will therapeutic cloning create immune matching?  It’s unclear.  At this point, we don’t even know if human embryonic stem cells are safe, let alone effective.  What’s important is that research be allowed to continue so we can find out.
    The bottom line: the race toward this potentially lucrative technology by states and other countries seems to be outpacing concerns about ethics, even though there is no evidence ES stem cells will cure anything (while adult stem cells already have plenty).  Now that they are on the verge of getting their way, the scientists are having one last twinge of conscience before charging full steam ahead.
1Erica Check, “Biologists seek consensus on guidelines for stem-cell research,” Nature 431, 885 (21 October 2004); doi:10.1038/431885a.
2Editorials: “California dreaming,” Nature 431, 723 (14 October 2004); doi:10.1038/431723a
3Constance Holden, “Bioethics: Stem Cell Researchers Mull Ideas for Self-Regulation,” Science, Vol 306, Issue 5696, 586, 22 October 2004, [DOI: 10.1126/science.306.5696.586].
If anyone should have a voice in the ethics of stem cell research, it should be Joni Eareckson Tada, the advocate for the disabled who has spent the last 37 years in a wheelchair herself.  She has done far more than the TV celebrities to help the afflicted.  Her organization “Joni and Friends” has supplied over 25,000 wheelchairs to the disabled poor in Africa and other third world countries.  Moreover, she could certainly be expected to look with hope over any therapies that might allow her to walk again.  Yet she remains a staunch opponent of embryonic stem cell research, for good reasons, as explained on the bioethics page of her website JoniAndFrends.org.
    Joni has appeared on radio talk shows and TV interviews, such as in a debate last week on Faith Under Fire.  The clarity of her logic is unimpeachable.  Yet it is unlikely that she can overcome the tear-jerking, emotional commercials by celebrity actors that tug at the heartstrings with empty promises that embryonic stem cells might cure your grandmother of Alzheimer’s or Parkinson’s disease, despite no track record and many problems (while adult stem cells are flourishing: for another example, see EurekAlert report this week about skin cells fighting brain tumors).  Meanwhile, beneficiaries of Prop. 71 stand to make a killing on taxpayer funds.  Follow the money trail: why don’t private investors support ES stem cell research?  Yet the taxpayers are going to have to foot the bill for a possible boondoggle that may take decades to show any results– maybe never, while a class of human beings will be created to be destroyed for scientific research (a good time to re-read John Durkin’s letter; see 09/03/2004 headline).  Since California voters never seem to find a bond issue they didn’t like, even when living in a state climbing out of near bankruptcy, the world is staged to see the next chapter in our brave new world opening on November 2.  Maybe the scientists will figure out how to be “ethical” while they’re laughing on the way to the bank.
Next headline on: Politics and Ethics.
Neo-Darwinism Falsified in the Lab   10/19/2004
Will the Spaniards be noted in history books as the ones who falsified neo-Darwinism?  Not likely; no one experiment would bring down a biological paradigm with such international and historical momentum behind it.  Nevertheless, looking at the results and conclusions of experiments by three evolutionary biologists at the Institut Cavanilles de Biodiversitat i Biologia Evolutiva, Universitat de Valencia in Spain, published in PNAS this week,1 it would be hard to find any support for the central tenets of neo-Darwinian theory: namely, that evolutionary adaptations arise by natural selection acting on beneficial mutations.  Instead, this paper shows experimental evidence that it doesn’t work.
    Neo-Darwinism, also termed the modern synthesis of evolutionary biology, was formulated in the 1940s to rescue Darwin’s views on natural selection from growing theoretical problems (see
07/02/2004 headline).  It incorporated the necessity of genetic mutations to provide the raw material for variation on which natural selection acts.  This revision was necessary when the rediscovery of Mendel’s laws of inheritance ruled out ideas of blending inheritance, showing instead that inherited characters were based on discrete entities (genes) that were passed on unaltered to the offspring.
    To test neo-Darwinian evolution in a microcosm, Rafael Sanjuán, Andrés Moya, and Santiago F. Elena worked with RNA viruses: organisms with a small, compact genomes that should respond quickly and noticeably to mutations.  The team was looking for epistatic interactions: i.e., the effects of multiple independent (non-allelic) mutations on each other, rather than the effects of single mutations alone.  These interactions can be antagonistic or synergistic: they can work against one another or with one another.  Epistasis is defined as “any interaction of nonallelic genes, especially the suppression by one gene of the effect of a nonallelic gene.”  Of note in this paper are the opening lines in the abstract that tell how rarely this important concept has been studied before (read: never):
The tendency for genetic architectures to exhibit epistasis among mutations plays a central role in the modern synthesis of evolutionary biology and in theoretical descriptions of many evolutionary processes.  Nevertheless, few studies unquestionably show whether, and how, mutations typically interact.  Beneficial mutations are especially difficult to identify because of their scarcity.  Consequently, epistasis among pairs of this important class of mutations has, to our knowledge, never before been explored.   (Emphasis added in all quotes.)
Let’s picture a 2x2 grid.  On the left side, label the rows “beneficial” and “deleterious.”  On the top, label the columns “synergistic” and “antagonistic.”  Now put two dots in each box, with the dots representing mutations that will interact with one another.  Quiz question: which box represents the only hope for evolutionary advancement?  Well, the bottom and right boxes are clearly not any help.  If the mutations are both deleterious and both antagonistic, at least they might turn each other off to stop the damage, like two criminals fighting each other instead of you.  If the mutations are both deleterious but synergistic, they will multiply each other’s damage, like two criminals ganging up on you.  If they are both beneficial but antagonistic, that won’t help, either, because it would be like two guardian angels having a squabble instead of helping you.  In short, neo-Darwinism’s only hope is to find mutations in the top left box: two good mutations that work synergistically, increasing your “fitness” in the evolutionary world of competition.  So how did the experiments go in the lab?
    They performed two classes of experiments to measure the effects of epistasis on mutations.  Continuing with the abstract, here is what they found:
Interactions among genome components should be of special relevance in compacted genomes such as those of RNA viruses.  To tackle these issues, we first generated 47 genotypes of vesicular stomatitis virus carrying pairs of nucleotide substitution mutations whose separated and combined deleterious effects on fitness were determined.  Several pairs exhibited significant interactions for fitness, including antagonistic and synergistic epistasis.  Synthetic lethals represented 50% of the latter.  In a second set of experiments, 15 genotypes carrying pairs of beneficial mutations were also created.  In this case, all significant interactions were antagonistic.  Our results show that the architecture of the fitness depends on complex interactions among genome components.
In other words, none of their pairs of mutations occupied the necessary box labeled “beneficial and synergistic.”  Half of the synergistic (working-together) actions they measured were “synthetic lethals” – which is like the two criminals both shooting the victim simultaneously.  The other 50% maybe didn’t kill the organisms but still decreased fitness overall.  The second experiment was all the more depressing: given two beneficial mutations in the same organism, all significant interactions were antagonistic.  This means the guardian angels were preventing each other from helping.  It recalls another paper in PNAS in March 2003 (see 03/17/2003 headline) that took into account indirect genetic effects, noting that increases in fitness do not act in isolation; they often counteract one another, creating “slippage on the treadmill.”
    In the current paper, the researchers found that beneficial mutations do not add up, even in the best of circumstances.  Neo-Darwinian theory assumes that beneficial mutations act independently, but the team found that of the eight actual best-case scenarios (two beneficial mutations working antagonistically, since none worked synergistically) over half decreased the total fitness of the result from what would be expected if the beneficial mutations acted alone.  They called this “decompensatory epistasis” if you need a new phrase to impress your friends at the water cooler.  What does this mean to neo-Darwinian theory?  “Indeed, when epistasis is decompensatory, both beneficial alleles involved in the interaction cannot spread to fixation in the population, because the double mutant is less fit than each single mutant.”  This drastically undercuts any hope for evolutionary progress.  Beneficial mutations are “scarce” to begin with, but more is not better – it’s worse.  Like adding hot sauce to ice cream, the benefits of each counteract one another when combined.  “As a consequence,” they continue, describing the only hope left, “lineages bearing alternative beneficial mutations should compete with each other on their way to fixation and, as a consequence of asexuality and clonal interference, only the best competitor will eventually become fixed in the population.”  That is, only one beneficial mutation can become fixed at a time, even in the best case scenario.
    The discussion of results in the paper by Sanjuán et al. hammers neo-Darwinian theory with additional gentle, but effective, blows.  First, they restate the basic finding: “Among pairs of deleterious mutations, although both synergistic and antagonistic epistases have been detected, interactions were predominantly antagonistic, such that their combined effect is significantly smaller than expected under a multiplicative model.”  And in the best-case scenario of artificially-induced beneficial mutations, “antagonistic epistasis represents the most abundant type of interaction among beneficial mutations, with several cases showing decompensatory epistasis.”
    How should these experiments impact evolutionary theory, including the “queen of evolutionary problems,” the origin of sex? (see 04/14/2003 headline).  Neo-Darwinists may well wish to run and hide:
The results reported here have two important implications for theories seeking explanations for the evolutionary advantage of recombination and sexual reproduction.  First, according to the Fisher–Muller argument, sex and recombination are advantageous because they combine into a common genotype beneficial mutations that arose in different ones, speeding up the rate of adaptationHowever, if the genetic architecture of RNA viruses determines that, in general, antagonistic epistasis and, in particular, decompensatory epistasis among beneficial mutations is the norm, then recombination would not necessarily imply a benefit in terms of adaptive evolution.  Second, sex might still be beneficial for RNA viruses as an efficient mechanism for purging deleterious mutations.  However, according with the Mutational Deterministic Hypothesis [i.e., the suggestion that sex enables a population to purge deleterious mutations from the genome], if this is the case, an excess of synergistic epistasis among deleterious mutations is required to compensate the 2-fold advantage of clonal reproduction [i.e., asexual reproduction].  Our first data set shows that synergistic interactions among random mutations are neither stronger nor more common than antagonistic interactions.  Indeed, the existence of variability among loci in the sign and strength of epistasis, and especially the dominance of antagonistic epistasis, decreases the parameter space over which sex may evolve.
Since the parameter space was small to begin with, their words sound euphemistic, as if to cheer up a prisoner facing a hanging at dawn that maybe someone will find an alibi: “Like who?  Like what?” the prisoner asks.  “I dunno; just supposin’,” the friend replies.  How sex may evolve: that’s somebody else’s problem.
    Also, they note, their results “impose a strong burden” on the “often invoked limitless adaptability of RNA viruses.”  Citing another paper, they quote, “RNA viruses might be more at the mercy of their mutation rates than we think.”  If decompensatory epistasis and antagonistic interactions are the general rules for mutations in all organisms, any hope for variability and adaptability due to mutation and selection has been severely limited, if not falsified, by these experiments.  On the contrary, they say their experiments demonstrate a mechanism for stability of the genome: “In this sense, because it involves masking the interaction among deleterious alleles, antagonistic epistasis might be seen as a sort of genetic mutational robustness.”  (See 09/22/2004 headline on robustness as a design constraint in the living cell.)
    In conclusion, they caution evolutionary modelers to realize that they can no longer merely assume fitness gains (if any) add up.  Mercifully, they use the words hint and suggest:  “Finally, we would like to hint that the above findings prompt the necessity of considering nonmultiplicative fitness effects in mathematical descriptions of viral evolution.”  Indeed, “the results we present here suggest that more realistic models must incorporate variance in the type and strength of epistasis among mutations.”  But did they themselves find any synergistic, beneficial epistatic effects by experiment?  None.  Maybe neo-Darwinism is like the businessman who lost money on every sale but thought he could make it up in volume.
1Rafael Sanjuán, Andrés Moya, and Santiago F. Elena, “Evolution: The contribution of epistasis to the architecture of fitness in an RNA virus,” Proceedings of the National Academy of Sciences USA, 10.1073/pnas.0404125101, Published online before print October 18, 2004.
Any scientific hypothesis must be testable and subject to falsification by experiment.  It is not enough to tell just-so stories, and describe things in glittering generalities with armchair scenarios.  Neo-Darwinian theory, the idea that natural selection acting on “scarce” beneficial mutations can produce all the diversity of life, from diving cormorants to catapulting chameleon tongues to sponge fiber optics to high-tech fruit fly aircraft to supersonic high-jumping froghoppers to efficient penguin, whale and dolphin flippers to fish physics students to glass-sculpturing diatoms to self-propelled motors, must be testable if it is to be declared scientific.
    So there.  These scientists finally put neo-Darwinism to the test in a microcosm that should have shown, if the principles were correct, a clear case of fitness increasing as a result of natural selection acting on beneficial mutations.  It failed.  It failed miserably.  Not only were no instances of synergistic beneficial mutations detected, the beneficial mutations that were artificially inserted worked against each other!  Neo-Darwinism is falsified!  And it was falsified not by creationists, but by evolutionary biologists working in the lab at an institute for the study of biological evolution!
    Now all we need to do is get the word out.  Stop the propaganda machine, stop the NCSE and ACLU threats at the school boards, stop the PBS NOVA programs and the media spin doctors.  Rearrange the museums, gather up the pro-Darwin displays and national park signs and toss them, along with neo-Darwinian theory itself, and by implication all of Charlie’s baggage that was already obsolete before 1940, onto the dumpster of discredited ideas.  Darwin’s Century is now a mere footnote of history, an unfortunate detour that killed 100 million people, but at least now we know better.
Next headline on: Darwinism and Evolutionary Theory.
National Geographic Faces Fact that Darwinism Is Minority View    10/18/2004
The cover of the November issue of
National Geographic is asking the question, “Was Darwin Wrong?  The work of the 19th-century English naturalist shocked society and revolutionized science.  How well has it withstood the test of time?”  The lead article by David Quammen notes that for decades, though evolution is supported by “overwhelming evidence,” 45% of Americans believe Darwinism had nothing to do with the origin of man, while an additional large share, about 37%, allow for theistic evolution.  This statistic has remained constant for two decades.  “The creationist conviction—that God alone, and not evolution, produced humans—has never drawn less than 44 percent,” Quammen notes with some surprise.  “In other words, nearly half the American populace prefers to believe that Charles Darwin was wrong where it mattered most.”
    Yet NG is not quite ready to capitulate; Stefan Lovgren writes for National Geographic News that evolution and religion can coexist, provided people are willing to give up a literal reading of the Bible. The Bible must be wrong, because “Scientific evidence shows that the universe was actually formed about 13.7 billion years ago” [see 01/02/2004 and 10/06/2004 headlines], “while the Earth was formed around 4.5 billion years ago,” Lovgren asserts [see 10/06/2004 and 09/20/2004 headlines].  “The first humans date back only a hundred thousand years or so” [see 09/29/2004 and 09/23/2004 headlines].  Both sides in the war of worldviews can get along, suggest some scientists, if religious people can just view evolution as God’s tool.
Sorry, appeasement will not work.  The King will not negotiate with unrepentant mythmakers.  He demands unconditional surrender.
Next headline on: Darwinism and Evolutionary Theory. • Next headline on: The Bible.
Darwinian Dogma Doubted: Cave Fish Go Blind on Purpose    10/18/2004
Contrary to previous belief, blind cave fish have the genes to build eyes but turn them off during development, reports
Science Now
When a body part is no longer needed, scientists usually assume that mutations accumulate in the genes controlling the structure, eventually preventing it from working or being made.  “That was the dogma,” says Stephen Ekker of the University of Minnesota, Twin Cities.  But because the cave fish eyes are actively killed, natural selection is probably doing its thing [sic], says Jeffery.  And that, he adds, might come as a surprise even to Darwin, who thought the cave fishes’ loss of sight might be an exception to the rules [sic] of natural selection.  The next step, of course, will be to figure out what the fish gain by losing their sight.   (Emphasis added in all quotes.)
So it’s not simply an issue of “use it or lose it,” the article states; “new research suggests that for some cave-dwelling fishes, blindness results from the careful coordination of gene expression, not simply from lack of use.”
    Science News also reported this story, describing how the researchers could induce blindness in sighted fish by controlling the expression of a gene, and could partially restore sight in blind fish with different gene regulation.  The article ends, “Because the genes orchestrate both mouth and eye development, the blind fish may have lost their eyes as t hey gained a more effective mouth—a useful feature for catching food in the dark.”
Creationists have argued that natural selection eliminates things, rather than constructing them, so this is no surprise.  What is surprising is that the eyes did not merely degenerate through disuse, and that the response could have occurred quickly through changes in the regulation of one gene during development.  Maybe there is a functional reason why this gene is being switched off in the darkness of the cave environment.  Maybe with eyes not in the way, it helps enhance the other senses the fish will need when sight is not possible.  Whatever the reason, natural selection cannot be credited with creating new function, because both eyes and mouths already functioned well. 
Next headline on: Darwinism and Evolutionary Theory. • Next headline on: Fish and Marine Life.
Planet-Building a Mess, or Theories a Mess?    10/18/2004
A news release from the
Spitzer Space Telescope operated by JPL says, “Astronomers Discover Planet Building is Big Mess.”  Data from the orbiting infrared observatory indicates that dust disks around stars appear to be dominated by collisions of large bodies.  Surprisingly, the dust disks do not correlate with the stars’ ages.  A study of 266 stars showed 71 with disks.  Contrary to the belief that disks condense into planets over time, some young stars showed no disks, and some old stars showed massive ones.  The disks appear to be subject to violent, swirling activity, if infrared signatures from these disks can be taken to indicate that collisions between large bodies are taking place now.  “Prior to these new results, astronomers thought planets were formed under less chaotic circumstances.”
    The project scientists are not worried, though, putting a positive spin on the “messy” findings: “Spitzer has opened a new door to the study of discs and planetary evolution,” said one, and another beamed, “These exciting new findings give us new insights into the process of planetary formation, a process that led to the birth of planet Earth and to life.”
    Astronomy Picture of the Day highlighted this story on October 19, stating that scientists expected to find dust disks depleting over time, but found the opposite.
Anyone see a solar system or planet or organism forming in the data?  The only thing Spitzer sees is heat from crashing bodies, not a process leading to planets and life.  “When embryonic planets [sic], the rocky cores of planets like Earth and Mars, crash together, they are believed to either merge into a bigger planet or splinter into pieces.”  There is indirect evidence for the latter, but the former is merely a belief. 
According to the most popular [sic] theory, rocky planets form somewhat like snowmen [sic].  They start out around young stars as tiny balls in a disc-shaped field of thick dust.  Then, through sticky interactions with other dust grains, they gradually accumulate more mass.  Eventually, mountain-sized bodies take shape, which further collide to make planets.  (Emphasis added in all quotes.)
Most people make snowmen by intelligent design.  When they have snowball fights, nothing creative emerges out of the mess – only pain for the impactee.  None of this evidence matched the evolutionary expectations: not the dates, not the timeline, not the formation of planets.  All Spitzer sees is colliding projectiles – no snowmen, no planets, no life.  Whaddya say we stick to the evidence, OK?
Next headline on: Stars. • Next headline on: Solar System. • Next headline on: Dating Methods.
If Mars Had Water, It Wasn’t for Long    10/18/2004
The Mars Exploration Rovers found evidence for the minerals jarosite and gypsum.  Jarosite has been found on earth in connection with lava and acidic, sulfur-rich fluids, but usually only persists in an arid environment, says a press release from
Virginia Tech based on a paper in Nature last week.1  Consequently, jarosite might be an indicator of a water-limited environment, and “liquid water may have been on Mars briefly.”
As to how much water was on Mars, the researchers do not know if there was a great deal for a short time or a little for a longer period.  However, they can say there was a geologically short window in which liquid water was present, suggesting there also was a limited time period when conditions may have been hospitable for life, [Donald] Rimstidt said.   (Emphasis added in all quotes.)

1Madden, Bodnar and Rimstidt, “Jarosite as an indicator of water-limited chemical weathering on Mars,” Nature 431, 821 - 823 (14 October 2004); doi:10.1038/nature02971.
The wet-Marsers are losing, and the Mars-lifers are down for the count.  The place reeks with sulfur and is bombarded by death rays.  The solar wind is dehydrating the whole planet.  Sorry, Percival.
Next headline on: Mars.
Stupid Evolution Quote of the Week    10/18/2004
This week’s entry has a little jargon in it, but if you remember what we’ve said about the tRNA synthetase family of proteins (see
05/26/2004, 07/21/2003 and 06/09/2003 headlines), you’ll get it.  Paul Schimmel and Karla Ewalt comment in Cell1 on new discoveries by Sampath et al.2 that two of these synthetases fuse together to regulate the inflammation reaction:
Aminoacyl tRNA synthetases are ancient [sic] proteins that appeared [sic] before the split [sic] of the tree of life [sic] into its three great kingdoms—archae [sic], eukarya, and bacteria.  The 20 enzymes—one for each amino acid—catalyze aminoacylation of tRNAs and thereby establish the rules of the genetic code by associating each amino acid with a nucleotide triplet (the anticodon of the tRNA).  The transition from the RNA world [sic] to the theater of proteins [sic] was thus made possible by the development [sic] of specific aminoacylation reactions.  While the central connection between synthetases and the code has long been recognized, the modern enzymes have surprised us with novel functions beyond aminoacylation.  They are key regulators and active components in a wide range of cellular functions from RNA splicing and transcription to apoptosis and angiogenesis....
    .... we now see that tRNA synthetases in mammalian cells have more diverse and comprehensive connections to the inflammatory system than was previously appreciated.  The diverse functions of these essential enzymes never cease to amaze!
  (Emphasis added in all quotes.)

1Paul Schimmel and Karla Ewalt, “Translation Silenced by Fused Pair of tRNA Synthetases,” Cell, Volume 119, Issue 2, 15 October 2004, Pages 147-148, doi:10.1016/j.cell.2004.10.001.
2Sampath et al., “Noncanonical Function of Glutamyl-Prolyl-tRNA Synthetase Gene-Specific Silencing of Translation,” Cell, Volume 119, Issue 2, 15 October 2004, Pages 195-208, doi:10.1016/j.cell.2004.09.030.
To be an evolutionist, you have to take the Crick brainwashing class.  This involves repeating the following quote by Francis Crick over and over until it is engraved on the heart with an iron stylus: “Biologists must constantly keep in mind that what they see was not designed, but evolved.”
Next headline on: Genes and DNA. • Next dumb story.
Ghost of Hitler Still Haunts Western Medicine    10/18/2004
“During the 1930s, the German medical establishment was admired as a world leader in innovative public health and medical research.  The question we want to examine is: ‘How could science be co-opted in such a way that doctors as healers evolved into killers and medical research became torture?’”  The question was posed by Dr. Alan Wells, medical ethics expert with the AMA, at a conference in Washington D.C. last week sponsored in conjunction with the Holocaust Museum, reports
EurekAlert.  He continued:
Many of the most important issues in medical ethics today – from genetic testing and stem cell research to caring for prisoners of war are directly affected by the experiences of medicine leading up to and during the Holocaust.  Physicians need to explore these issues without getting caught up in political agendas or the results can be something we never intended and cause great harm.   (Emphasis added in all quotes.)
He recounts that German doctors were considered leaders in medical innovation in the years leading up to the Holocaust.  Yet their efforts were aimed by the Reich at improving the purity of the Aryan race.  This meant the unfit or non-Aryan were viewed as threats to health:
Adolf Hitler spoke of Germany as a body with himself as the doctor.  He wanted to make Germany ‘healthy’ by eliminating diseased, unhealthy [sic] parts of the body.  At first this meant killing the disabled.  But because the Nazis also believed that Jews possessed ‘bad’ genes, they, too, came to be portrayed by public health ‘experts’ and ‘scientists’ as a threat to racial purity and a healthy nation.
According to Dr. Patricia Heberer of the Holocaust Museum, the evil actions grew out of eugenics, a “distortion” of Charles Darwin’s theories of survival of the fittest.  The abuses in Nazi Germany continue to influence medical practice today, the article states.  For instance, Dr. Wells says, “our codes of ethics demand that we treat every person equally, without regard to race or ethnic background.  This ethical obligation is a direct outgrowth of the horrors of Nazi medicine.”  He cautions that even though these horrors seem so long ago, we can never forget this history.  See also the 07/30/2001 and 04/22/2004 headlines.
A grave cause for alarm is that people are forgetting.  First of all, let’s clear up the distortion that eugenics was a distortion of Darwin’s theories.  The subtitle of Charlie’s book was the preservation of favoured races in the struggle for life.  What does “favoured races” mean when speaking of the human race?  In later editions of his book, Darwin moved from the term “natural selection” to Herbert Spencer’s phrase “survival of the fittest.” The Victorian British were caught up in the myth of progress, and many, including Darwin and his friends, held racist beliefs, some of them radical.  The success of their empire surely proved they were the fittest, did it not?  The “father of eugenics” was Darwin’s own cousin, Francis Galton.  Ernst Haeckel took the core beliefs of survival of the fittest and eugenics to Germany, where they were taking hold before Hitler came to power.  Hitler merely lifted constraints on trends that were already established, the article says:
Some eugenics programs, such as laws sanctioning the sterilization of the ‘feeble minded’ [sic], initially met with resistance throughout the world, including in Germany.  But when the Nazis came to power, and particularly during World War II, these constraints disappeared as the Nazi regime was able to implement its radical version of medicine.
Lest anyone think the evil was constrained to the German borders, eugenics and anti-Semitism was widespread throughout Europe and America at the time; America itself had a pre-Hitlerian forced sterilization program (see 10/21/2001 headline).  There is a direct line philosophically from the ideas of Darwin to the actions of Nazi Germany, as historian Richard Weikart has documented.
    Nazi Germany provides a classic case study that bad ideas can have horrific consequences, and that “good” doctors and scientists can be hoodwinked into letting their talents be co-opted for evil.  How could we forget?  Yet that is exactly what Nature suggested last month (see 09/27/2004 headline), that we need to get over our ethical hangups stemming from Nazi abuses, and move forward with today’s medical technologies.  Notice how pre-Hitler eugenics started with killing the disabled.  Sound familiar?  (Clue: Terry Schiavo.)  Hitlerian medicine also justified killing of those that it defined as not really human (clue: human embryos).  Peter Singer has advocated killing senior citizens and children based on Darwinian principles.  Now we are on the verge of synthetic biology, whose horrors can only be imagined (see 10/11/2004 headline).  Hitler may be gone, but the core beliefs of Darwinism still reign supreme in the halls of academia.  Another holocaust may await a new madman rising to power and promising Big Science all it wants, as he steers it to his agenda.
    Welcome to 2004.  On one side we have radical Muslims wanting to disconnect our heads and nuke our cities.  On the other we have Big Science ready to endorse the New Eugenics.  If we don’t want evil to triumph, doing nothing is not an option.  As J. Gresham Machen warned in the pre-Nazi, pro-eugenics days of 1913, “What is today matter of academic speculation begins tomorrow to move armies and pull down empires.  In that second stage, it has gone too far to be combatted; the time to stop it was when it was still a matter of impassionate debate.”  Will the archival footage of World War II help us learn from history this time?
Next headline on: Politics and Ethics. • Next headline on: Health.
Grand Canyon Creation Book Stays on Shelves    10/14/2004
The ruckus over a creation-oriented geology book on Grand Canyon at the Visitor Center (see
01/18/2004 headline) is back in the news.  The Environmental Media Services reports that plans for a review have been shelved by the park:
Despite telling members of Congress and the public that the legality and appropriateness of the National Park Service offering a creationist book for sale at Grand Canyon museums and bookstores was “under review at the national level by several offices,” no such review took place, according to materials obtained by PEER under the Freedom of Information Act.  Instead, the real agency position was expressed by NPS spokesperson Elaine Sevy as quoted in the Baptist Press News:
    “Now that the book has become quite popular, we don’t want to remove it.”
  (Emphasis added in all quotes.)
The group calling itself “Public Employees for Environmental Responsibility” (PEER) complains that the Bush Administration has a “Faith-Based Parks” agenda:
“Promoting creationism in our national parks is just as wrong as promoting it in our public schools,” stated PEER Executive Director Jeff Ruch, “If the Bush Administration is using public resources for pandering to Christian fundamentalists, it should at least have the decency to tell the truth about it.”
This reporter found copies on the bottom row of the North Rim Visitor Center in August.  The Ranger on duty indicated that people seem to like the book and want it, so they keep it on hand.
Update  On Oct. 19, World Net Daily posted an article on the controversy.  A National Park Service official said the review has not been shelved, but is “being looked at very carefully because it could be precedent-setting throughout the Park Service.”
The Darwin Party can only thrive in a totalitarian regime.  They are so threatened by other interpretations of the scientific evidence, they must resort to intimidation, the courts, and lies to try to stop the other side from being heard.  The book in question is not a flaky mythology like some native American stories that get evangelistic support at many national parks.  Grand Canyon: A Different View was written by scientists (at least 13 with PhDs) and canyon explorers with a lot of experience studying the canyon.  They just don’t subscribe to the reigning uniformitarian myth, but neither do some non-creationist geologists (see 07/22/2002 headline).  Many of the evidences supporting the Flood model are well known within the scientific community.  The secularists have no explanation for these things, such as 160 million years of missing strata with no evidence of erosion, and all they can offer is just-so stories that assume the ages anyway.  It is perfectly legal for any scientific viewpoint to be heard, especially one that explains these things in a superior fashion.  Why should it be “legal” and “appropriate” for the Bush Administration to “pander” to the atheists?  Promoting atheism in our national parks is just as wrong as promoting it in our public schools.  PEER should at least have the decency to tell the truth about it.  Buy the book AT the Grand Canyon visitor center if you can.  It’s not only a vote for fairness, it’s a worthwhile book to read and enjoy.
Next headline on: Geology. • Next headline on: Politics and Ethics.
How a Darwinist Explains “Living Fossils”    10/13/2004
Darwinism is a flexible concept that must embrace a wide variety of observations, from apparently fast-evolving plants (see
10/12/2004 item on maize) to organisms that seem to remain unevolved for eons.2  Darwin himself saw this flexibility as a strength for his unifying concept of common descent; others criticized it as rationalization (i.e., a concept that can explain anything explains nothing).  Take the case of so-called “living fossils,” organisms whose modern counterparts are virtually identical to fossils sometimes hundreds of millions of years old.  If a land animal could evolve into a whale in 50 million years, for instance, why would a horseshoe crab show no change at all for 10 times as long, 500 million years? (see 06/21/2002 headline).  How can the fluidity of constant evolutionary change over time be reconciled with observations that many different kinds of creatures – trees, salamanders, ostracods, reptiles, insects and fish – have apparently not evolved at all?
    This subject was recently tackled by Lee Hsiang Liow of the Committee on Evolutionary Biology at the University of Chicago.  Examining fossil crinoids (branch-like echinoderms that attach to the seafloor, also called sea lilies and feather stars), Liow tested “Simpson’s Rule” of “the survival of the relatively unspecialized,” a rule George Gaylord Simpson proposed in 1944 as an explanation for living fossils and long-lived taxa.  Basically, it suggests that specialists evolve, but generalists persist.  Liow applied the rule to crinoids and published the results in American Naturalist.1  She studied 1,195 species, representing 752 genera, and concluded that long-lived crinoid genera seem relatively unspecialized, in accordance with Simpson’s Rule, but the reverse is true for higher taxa.  The conclusion seems to raise doubts about the utility of Darwinian explanations.
Prolonged stasis in a world of change is a puzzling biological phenomenon.  Extremely long-lived or geologically long-ranging taxa have been a popular subject of discussion for paleontologists and neontologists alike ever since Darwin ([1859] 1964) coined the term “living fossils.”....
  (Emphasis added in all quotes.)
Whether it is called bradytely, arrested evolution, morphological stasis, long-lived taxa or something else, or whether “living fossils” are dubbed paradoxical, relictual, primitive or specialized, the phenomenon of stasis has rarely been studied in a quantitative manner.  This Liow set out to correct, at least in the case of crinoid evolution.  The crinoid class is ideal for study because it span much of the geological column and contains many well-characterized examples, both fossil and living.  She compared samples and deduced a morphological average, then tried to determine if longevity was a function of “bizarreness.” 
Simpson implicitly took a comparative approach when he wrote about the “rule of the survival of the relatively unspecialized” (1944, p. 143).  He thought that unspecialized subgroups of a clade seem to persist for longer periods of geologic time but did not explicitly define “specialization.”  Here, I quantify specialization by comparing individual morphologies to a group mean; the closer a morphology is to a group mean, the less specialized it is.  I ask whether long-lived genera ... in any given crinoid order occupy regions of morphospace that are random with respect to the mean morphology of that order.  Could survival be correlated with morphological bizarreness or a deviant morphology ...?  Or would long-lived genera have morphologies close to the mean morphology...?
The short answer is: Simpson seems to be right on one level.  “I find that the morphologies of long-lived crinoid genera are, in general, closer to mean morphologies than shorter-lived genera in the same order.”  But when higher taxonomic categories are examined, the rule fails:
Similarly, but from a completely different conceptual perspective, I ask whether long-lived crinoid genera in any given crinoid higher taxon (e.g., suborder, order) occupy regions of morphospace that are random with respect to a basal morphology of that higher taxon.  I find that mean morphological distances of long-lived genera from basal morphologies are seldom distinct from those of their shorter-lived relatives.
In other words, she found a contradiction in the trend between lower and higher taxonomic groups.  Part of the problem is the fuzziness of the evolutionary record:
There is no available phylogenetic framework for comparing rates of character transformation in the global pool of fossil crinoids.  Likewise, there are no detailed samples of crinoid lineages in a stratigraphic column for investigating character reversals, convergence, or the lack thereof.
Nevertheless, she found a way to compare features: “The morphological characters used here are not assumed to be strictly homologous but are assumed to reflect only general fossilizable morphology determined consistently within the crinoid bauplan [body plan].”  Also, the fossil history was determined from location in the geological column, both first and last appearance (see 05/21/2004 headline), and when dealing with fossils, the classification into genus and species is not always clear cut.  Geological history adds to the confusion:
Just as in previous analyses when genera are grouped according to orders, genera in each period are mostly short lived.  However, rarefied samples of shorter-lived genera through each period inform us that the long-lived taxa can be more, less, or equally deviant compared with shorter-lived taxa of an equivalent sample size (table C2).
    Genera that are extremely long lived within each order are also more likely ... than other genera in the database to have passed through one or more mass extinctions even though passing through mass extinctions does not necessarily ensure persistence.
I.e., the longer you live, the more dangers you have faced, but facing dangers doesn’t make you Supercrinoid.  She claims, nevertheless, that “the likelihood of the occurrence of ‘living fossils’ or long-lived taxa increases with time,” a truism given the evolutionary and geological-time assumptions.
    Liow addresses more factors that confuse the picture and could bias the results, such as taxonomic lumping, limited range of some species, the tendency for long-lived species to swamp short-lived ones, “issues of stratigraphic resolution of age dating,” and disagreement over how to define a “long-lived” taxon.  This discussion seems intended to cushion the next paragraph, the conclusions.  Before opening the curtain, she cautions, “In summary, longevity is relative and dependent on taxonomic inclusiveness.  These important axioms are often neglected in articles that address extreme persistence or morphological conservatism.”
    Conclusion time.  What can be claimed based on this detailed analysis of crinoids?
  1. “First, most taxa (genera and families) are short lived and ‘average”.... which implies that experiments in morphology [sic] are usually not long lived.”  This seems to suggest Mother Nature is a bumbling tinkerer.
  2. “Second, long-lived genera within orders are often less morphologically deviant or less specialized than expected when compared with rarefied samples of corresponding shorter-lived genera.”  This was apparently a big surprise.  “In other words, long-lived genera are not only not unusual, some are unusually average!” (exclamation in the original).
  3. “Third, patterns of morphological deviations from basal morphologies versus durations are unclear.”  Enough said.
  4. Fourth, there appears to be an increase in long-lived groups through time, but this appears to be an artifact of the assumptions of phylogeny and geologic ages.
  5. “Fifth, taxonomic ranks and inclusiveness of higher taxa are critical factors when discussing longevity because identities of long-lived taxa may dramatically change according to these factors.”  This suggests that previous investigators failed to see the big picture.
  6. “Last, identities of long-lived taxa may change with respect to which definitions of longevity are used.  This may or may not (as was the case for this article) change conclusions being drawn on long-lived taxa.”  This seems to say that conclusions are a function of definitions, not data.
She lists a few suggestions that have been offered for why some species persist.  Maybe “extinctions are not biologically random,” for instance.  Overall, though, she is confident that “Based on the results of the current study, I rule out the idea that long-lived genera are morphologically